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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAsrYMVIPhAEkk9VHbftnkMrsqIoJBumiNB-m9U-D6AUU#_6 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAsrYMVIPhAEkk9VHbftnkMrsqIoJBumiNB-m9U-D6AUU#_5 http://www.w3.org/ns/prov#value Furthermore, amplification and overexpression of SRC-3 were observed in 10% and 64% of human primary breast cancers, respectively [18]. This observation indicates that SRC-3 is not only essential for the normal mammary development, but also plays a role in breast tumorigenesis It has been demonstrated by transient transfection assay that RPF1 and E6-AP could potentiate the ligand-dependent transcriptional activity of the PR, the glucocorticoid receptor, and other nuclear receptors [19,20]. 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