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[Using a tumor necrosis factor (TNF)-?-induced bone marrow (BM)-MSC injury model in vitro and a rat MI model in vivo, we showed in the current study that miR-23a was involved in TNF-?-induced BM-MSC apoptosis through regulating caspase-7 and that the injection of BM-MSCs overexpressing miR-23a could improve left ventricular (LV) function and reduce infarct size in the rat MI model.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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