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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAuLW8ca6ApMjHhDUw43exiXYwa4h-aIyccdq6jCiqRtw#_6 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAuLW8ca6ApMjHhDUw43exiXYwa4h-aIyccdq6jCiqRtw#_5 http://www.w3.org/ns/prov#value Conversely, because the TIF1? antisense-ribozymes had no effect on GAPDH mRNA levels (Fig. 1D, bottom panel), they appear to be specific for the TIF1? mRNAs. These data show that the TIF1? antisense-ribozymes specifically ablate expression of endogenous TIF1? mRNA. As seen in Figure 2B, CD11b, CD11c, and CD14 levels increased in response to PMA-treatment of U937 clones expressing the ?-gal antisense-ribozyme. 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