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All rights reserved. http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/dc/elements/1.1/title BEL Framework Large Corpus Document http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/authoredBy http://www.tkuhn.ch/bel2nanopub/RAudCk4q1AfelCywIYwxvTT1YNN68ZAz2Lg_6BO5Hkr_0#_6 http://resource.belframework.org/belframework/1.0/knowledge/large_corpus.bel http://purl.org/pav/version 1.4 http://www.tkuhn.ch/bel2nanopub/RAudCk4q1AfelCywIYwxvTT1YNN68ZAz2Lg_6BO5Hkr_0#_5 http://www.w3.org/ns/prov#value Kang et al. (1999) demonstrated that the ARA54 gene (RNF14; 605675) can function as a coactivator for androgen-dependent transcription on both wildtype and mutant androgen receptor. They found that in the presence of a certain amount of 17-beta-estradiol or hydroxyflutamide, the transcriptional activity of a specific AR mutant was significantly enhanced, whereas that of wildtype and another AR mutant was not. They therefore suggested that both ARA54 and the positions of the AR mutation might contribute to the specificity of AR-mediated transactivation. 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