@prefix this: . @prefix sub: . @prefix beldoc: . @prefix rdfs: . @prefix rdf: . @prefix xsd: . @prefix dct: . @prefix dce: . @prefix pav: . @prefix np: . @prefix belv: . @prefix prov: . @prefix Protein: . @prefix entrez: . @prefix geneProductOf: . @prefix go: . @prefix rgd: . @prefix hasAgent: . @prefix mesh: . @prefix occursIn: . @prefix species: . @prefix pubmed: . @prefix orcid: . sub:Head { this: np:hasAssertion sub:assertion; np:hasProvenance sub:provenance; np:hasPublicationInfo sub:pubinfo; a np:Nanopublication . } sub:assertion { sub:_1 geneProductOf: entrez:24179; a Protein: . sub:_2 hasAgent: sub:_3; a go:0003824 . sub:_3 geneProductOf: rgd:69410; a Protein: . sub:_4 occursIn: mesh:D007668, species:10116; rdf:object sub:_2; rdf:predicate belv:increases; rdf:subject sub:_1; a rdf:Statement . sub:assertion rdfs:label "p(EGID:24179) -> cat(p(RGD:Asah2))" . } sub:provenance { beldoc: dce:description "Approximately 61,000 statements."; dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved."; dce:title "BEL Framework Large Corpus Document"; pav:authoredBy sub:_6; pav:version "1.4" . sub:_5 prov:value "Furthermore, we tested the physiological activators of PKC, like platelet-derived growth factor (PDGF-BB), angiotensin II, and the stable ATP analog ATPgamma S, which all evoke phosphoinositide hydrolysis and generation of 1,2-diacylglycerol, the endogenous activator of PKC (24-26). All these substances mimicked the effect of TPA and reversed the NO-mediated inhibition of neutral ceramidase activity"; prov:wasQuotedFrom pubmed:12359735 . sub:_6 rdfs:label "Selventa" . sub:assertion prov:hadPrimarySource pubmed:12359735; prov:wasDerivedFrom beldoc:, sub:_5 . } sub:pubinfo { this: dct:created "2014-07-03T14:30:06.232+02:00"^^xsd:dateTime; pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 . }