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Approximately 61,000 statements.
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Furthermore, we tested the physiological activators of PKC, like platelet-derived growth factor (PDGF-BB), angiotensin II, and the stable ATP analog ATPgamma S, which all evoke phosphoinositide hydrolysis and generation of 1,2-diacylglycerol, the endogenous activator of PKC (24-26). All these substances mimicked the effect of TPA and reversed the NO-mediated inhibition of neutral ceramidase activity
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