@prefix dct: .
@prefix this: .
@prefix sub: .
@prefix beldoc: .
@prefix rdfs: .
@prefix rdf: .
@prefix xsd: .
@prefix dce: .
@prefix pav: .
@prefix np: .
@prefix belv: .
@prefix prov: .
@prefix chebi: .
@prefix go: .
@prefix Protein: .
@prefix mgi: .
@prefix geneProductOf: .
@prefix hasAgent: .
@prefix species: .
@prefix occursIn: .
@prefix pubmed: .
@prefix orcid: .
sub:Head {
this: np:hasAssertion sub:assertion;
np:hasProvenance sub:provenance;
np:hasPublicationInfo sub:pubinfo;
a np:Nanopublication .
}
sub:assertion {
sub:_1 hasAgent: sub:_2;
a go:0042789 .
sub:_2 geneProductOf: mgi:101898;
a Protein: .
sub:_3 occursIn: species:10090;
rdf:object sub:_1;
rdf:predicate belv:decreases;
rdf:subject chebi:46024;
a rdf:Statement .
sub:assertion rdfs:label "a(CHEBI:\"trichostatin A\") -| tscript(p(MGI:Pou2f1))" .
}
sub:provenance {
beldoc: dce:description "Approximately 61,000 statements.";
dce:rights "Copyright (c) 2011-2012, Selventa. All rights reserved.";
dce:title "BEL Framework Large Corpus Document";
pav:authoredBy sub:_5;
pav:version "1.4" .
sub:_4 prov:value "Hdac inhibitors disrupt Oct1 binding to the octamer element Oct1 interacts with the H2A octamer element with a high efficiency and represses Ifng-inducible H2A promtoer activation in Rb-defective tumor cell lines the reduction in binding activity seen for Oct1 was specific, as 2 other proteins, Nfy and Yy1, were not affected in their ability to bind DNA following treatment of the cells with Hdac inhibitors";
prov:wasQuotedFrom pubmed:11533238 .
sub:_5 rdfs:label "Selventa" .
sub:assertion prov:hadPrimarySource pubmed:11533238;
prov:wasDerivedFrom beldoc:, sub:_4 .
}
sub:pubinfo {
this: dct:created "2014-07-03T14:29:58.051+02:00"^^xsd:dateTime;
pav:createdBy orcid:0000-0001-6818-334X, orcid:0000-0002-1267-0234 .
}