sub:provenance {
  beldoc: dce:description "Approximately 2000 hand curated statements drawn from 57 PubMeds." ;
    dce:rights "Copyright (c) 2011-2012, Selventa. All Rights Reserved." ;
    dce:title "BEL Framework Small Corpus Document" ;
    dc:license "Creative Commons Attribution-Non-Commercial-ShareAlike 3.0 Unported License" ;
    pav:authoredBy sub:_7 ;
    pav:version "1.6" .
  sub:_6 prov:value """To test whether the cell proliferation defect observed with Rin1 depletion was because
of altered EGFR signaling, A549 cells were transfected with control or Rin1-specific
morpholinos, serum-starved, and then stimulated with EGF. The levels
of phosphorylated ERK1/2 (pERK) were used to assess EGF signaling (Fig. 2C). Depletion
of Rin1 from A549 cells resulted in at least a 2-fold decrease in pERK levels
(Fig. 2, C and D), but not total ERK levels (supplemental Fig. 2), indicating
that the decrease in cell proliferation correlated with a decrease in EGF signaling.""" ;
    prov:wasQuotedFrom pubmed:19570984 .
  sub:_7 rdfs:comment "support@belframework.org" ;
    rdfs:label "Selventa" .
  sub:assertion prov:hadPrimarySource pubmed:19570984 ;
    prov:wasDerivedFrom beldoc: , sub:_6 .
}