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[Given the dependence of ?-aminobutyric acid type A (GABAA) receptor subunit functioning on localized calcium currents, and the functional link between GABAergic inhibition and ataxia, we hypothesized that cerebellar GABAA receptor expression is differentially affected in Cacna1a mutants and contributes to the ataxic phenotype.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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